Sorghum is a staple food in African, Asian, and South American diets. In the U.S. it is typically found in animal feed or made into ethanol.
But sorghum is becoming a popular food item in American diets. It contains 10 grams of protein per half cup serving. It is also a good source of fiber, antioxidants and is gluten-free. It is the latter that has landed sorghum into many American diets.
Using sorghum in gluten-free foods has helped those with Celiac disease or other medically diagnosed reasons to avoid gluten. It helps increase whole grain consumption in gluten-free diets.
Sorghum also decreases insulin and glycemic responses compared to corn and rice. While sorghum has a lower glycemic index than wheat, sorghum syrup does not.
Antioxidants in sorghum help prevent cell and DNA damage. Many studies have shown the potential of sorghum to decrease certain cancer risks. While it may not be the cure to cancer, it certainly is a healthy addition to the diet.
Toss cooked sorghum into soups or salads, use in place of oatmeal for breakfast, or pop it like popcorn!
Source: www.foodinsight.org/sorghum-gluten-free-ancient-grain-fiber-antioxidants
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References:
ansgildied.com
AAS shouldn’t be confused with different forms of steroids corresponding
to corticosteroids (e.g., cortisone or prednisone), which haven’t
any anabolic effects and therefore little abuse potential (Pope and Brower, 2005; Sheffield-Moore
and City, 2004). When taken in supraphysiologic doses, AAS allow users to tremendously increase muscle power and athletic
performance, usually nicely past the limit attainable by pure means (Kouri et al., 1995).
The nice majority of illicit AAS customers, nevertheless,
aren’t elite athletes; certainly many usually are not competitive athletes
at all, however merely individuals who want to become more
muscular (Buckley et al., 1988; Kanayama et al., 2001b; Parkinson and Evans, 2006).
Atrophy happens due to a number of elements together with age, disease, and reductions in anabolic stimuli, including unloading due
to circumstances similar to microgravity, immobilization, or spinal cord injury (2, 21, 38,
40). These situations typically act through the ubiquitin-proteasome system,
the Ca++-dependent calpains, and the lysosomal cathepsins pathway, which in combination lead to muscle protein breakdown and eventual atrophy (3).
Conditions of atrophy are marked by elevated transcription and expression of
genes corresponding to atrogin1 and MuRF-1 (39).
In addition, AAS have virilizing results, which clearly isn’t
an issue in males however has great scientific significance in girls.
These results embody dysphonia or deepening of the voice, hirsutism and clitoromegaly.
Among AAS customers there is the assumption that AAS might
trigger gynecomastia through alternative pathways, such as elevated progestin action on the mammary
glands or elevated prolactin ranges. Whereas gynecomastia can develop in patients with hyperprolactinemia, the condition arises secondary to
the gonadotropin suppression prolactin may cause (203).
For females taking testosterone for superior breast most cancers, unwanted effects are generally associated to masculinization—voice
deepening, clitoral enlargement, and hirsutism. Testosterone is answerable for the growth and growth
of male sex organs and the secondary intercourse traits such as voice deepening, hair distribution, and redistribution of muscle and fats.
It also contributes to bone growth, pink blood cell production, decreased excretion of calcium in the urine,
and the retention of sodium, potassium, and phosphorous.
Any contribution from exogenous DHEA or androstenedione might be largely moderated by the big quantity of testosterone contributed by the testis.
In girls, an increase in efficiency may be potential following ingestion of those supplements, as
circulating testosterone can be expected to extend.
The plasma focus of endogenous testosterone is approximately 1/10th that found in men and the relative proportion arising from peripheral conversion of weaker androgens is much
larger.
A recent research evaluated the liver results of 5
weeks of administration of ND in rats. The outcomes highlighted an increase of
plasma levels of liver necrosis markers, a rise in collagen deposition in liver
parenchyma, portal space, and the centrolobular vein [84].
The mechanism involved in collagen deposition could possibly be the increase within the number and in the activity of Kuppfer
cells. In this regard, Kuppfer cell activation leads
to the production of many inflammatory cytokines corresponding to TGF-b1, NF-Kb,
IL-1b, associated to the liver fibrosis process
[85,86].
In all medical cases, excluding cancer, AASs have shown efficacy in weight achieve.
Extra importantly, remedy with anabolic steroids is linked to a
particular form of acute cholestasis also recognized as “bland cholestasis”.
The liver damage typically arises within 1 to four months of beginning therapy, but may
be delayed to as long as 6 to 24 months (Case 1).
The participant might press a button to build up points exchangeable for money (the “non-aggressive”
response) or press one other button to subtract factors
from his opponent (the aggressive response). In reality, the unseen “opponent” was a computer program that intentionally subtracted factors from the participant to find a way to provoke an aggressive response.
In this protocol, participants displayed strikingly and considerably higher
levels of aggression when receiving 600 milligrams per week
of testosterone as compared to placebo. Anabolic androgenic steroid (AAS) and performance-enhancing drug
(PED) use is a prevalent medical concern, especially among men, with an estimated 2.9–4 million People utilizing AAS of their lifetime.
Prior studies of AAS use reveal an association with polycythemia, dyslipidemia, infertility,
hypertension, left ventricular hypertrophy, and a quantity of
behavioral disorders.
Nowadays, except you are prescribed testosterone (often as testosterone
replacement therapy), steroids are illegal to buy and sell.
If you compete in sports, detection of steroids means a protracted ban from competitors.
Additionally, varied research have reported a rise in signs of schizophrenia,
homicidal tendencies and psychosis [2]. When you place a load of exogenous testosterone into your physique,
you possibly can surpass your regular ranges of T.
The dose is what’s called supraphysiological, as
a outcome of it may possibly ramp up T levels method above what your hypothalamus regulates as an higher restrict.
If you’re a man and your T ranges drop under 300 ng.dL your doctor will
diagnose you with a condition called hypogonadism
– clinically low testosterone levels.
References:
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